The fructose survival hypothesis views obesity as a low-energy state. Identifying fructose as the conduit that redirects active energy replacement to fat storage shows that fructose is what drives energy imbalance, which unites theories.
While more work is needed to fully validate this unifying hypothesis, this is a hopeful first step in potentially identifying more targeted preventions for obesity and related metabolic imbalance management.
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Obesity as a low-energy state, induced and extended by fructose.
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